Mice with an SOD1 mutation and an ALS-like disease survived longer after researchers blocked the immune-system protein interleukin-1-beta.

Posted on Thursday, August 12, 2010 - 06:42, By: Margaret Wahl
Elevated levels of an immune-system protein called interleukin-1-beta (IL-1-beta) exacerbates a disease in mice that closely resembles human amyotrophic lateral sclerosis (ALS), and blocking this protein extends survival in these mice, a research team in Germany has found. The study adds to the...